Role of Zinc in Brain Homeostasis and Neurological Disorders
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cells of the Nervous System".
Deadline for manuscript submissions: 30 June 2025 | Viewed by 902
Special Issue Editor
2. Department of Psychology, University of Calgary, Calgary, AB T2N 1N4, Canada
3. Department of Cell Biology and Anatomy, University of Calgary, Calgary, AB T2N 1N4, Canada
4. President-Elect, International Society for Zinc Biology
Interests: zinc neurobiology; experience-dependent plasticity; adult neurogenesis; brain development; brain injury
Special Issue Information
Dear Colleagues,
That zinc is a critical factor in the health and development of the nervous system has long been recognized. Zinc is an essential contributor to normal cellular function through its roles as a cofactor for many enzymes, as a vital component of protein structures, and as an intracellular and extracellular signaling ion. The importance of maintaining zinc homeostasis is evidenced by the 24 zinc-transporting proteins that have evolved to transport zinc into and out of cells and between various cellular compartments, with altered levels of zinc being implicated in numerous disorders, including Alzheimer’s disease, autism, schizophrenia and depression.
This Special Issue will examine the role of zinc in the normal adult and developing brain and how zinc dyshomeostasis can lead to brain dysfunction.
Prof. Dr. Richard H. Dyck
Guest Editor
Manuscript Submission Information
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Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- zinc
- neurotransmitter
- neuromodulator
- homeostasis
- zinc transporters
- ZnTs
- Slc30
- ZIPs
- Slc39
