Advance in Ca2+ Homeostasis
A special issue of Cells (ISSN 2073-4409).
Deadline for manuscript submissions: 20 October 2024 | Viewed by 129
Special Issue Editor
Interests: Ca2+ homeostasis; excitation–contraction coupling; cardiac muscle; skeletal muscle; B lymphocyte; ryanodine receptors; TRP channels; reticulum–mitochondria contact sites
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Calcium ions are the major second cell messenger and control a plethora of biological mechanisms, including muscle contraction. Their homeostasis is orchestrated like a musical score where the slightest abnormality or dysfunction of one of the partners (pumps, channels, exchangers, binding proteins, etc.) or the slightest change in the spatio-temporal properties of the signal can lead to a physiological process to a pathological situation in the long term, i.e., as a cell cacophony.
The aim of this Special Issue is to bring together original data and reviews on the Ca2+ role in skeletal and cardiac muscles. Contributions that address any determinant in the fine tuning of Ca2+ handling, involved in the mechanisms of excitation–contraction coupling, muscle function, plasticity or else in muscular and cardiovascular diseases, are welcome.
Dr. Sylvie Ducreux
Guest Editor
Manuscript Submission Information
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Keywords
- calcium homeostasis
- skeletal muscle
- cardiac muscle
- excitation–contraction coupling
- calcium channel
- calcium pumps and exchangers
- mitochondria and calcium
- sarcoplasmic reticulum and calcium
- calcium binding protein
- calcium signaling
- muscular disease
- cardiovascular disease
