The Role of Oxidative Stress and Inflammatory Cytokines in Glomerular Diseases
A special issue of Biology (ISSN 2079-7737). This special issue belongs to the section "Genetics and Genomics".
Deadline for manuscript submissions: 30 November 2024 | Viewed by 111
Special Issue Editor
Special Issue Information
Dear Colleagues,
Impaired kidney function is now considered to be an independent risk factor for cardiovascular disease. Therefore, current studies are focused on preserving kidney function, as well as preventing the progression of impaired kidney function to end-stage renal disease (ESRD). It is recognized that glomerular disease is a common cause of impaired kidney function, and that reduced glomerular filtration can lead to ESRD. Data also indicate that oxidative stress and inflammatory cytokines promote the progression of glomerular disease to ESRD. Thus, the aim of this Special Issue is to publish original research articles and reviews that inform about the contributions of genomic and genetic mechanisms that both preserve glomerular cell function and prevent glomerular disease progression to ESRD by processes regulating oxidative stress and inflammatory cytokine production. Submissions utilizing advanced in vitro and in vivo research approaches are particularly encouraged.
Prof. Dr. Ahmed Abdelrazik Elmarakby
Guest Editor
Manuscript Submission Information
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Keywords
- sex differences
- glomerular disease
- end-stage renal disease
- oxidative stress
- inflammation and fibrosis
