Cardiovascular Disease of Animals: Pathophysiology, Anatomy, Diagnosis and Treatment

A special issue of Applied Sciences (ISSN 2076-3417). This special issue belongs to the section "Applied Biosciences and Bioengineering".

Deadline for manuscript submissions: 30 June 2024 | Viewed by 932

Special Issue Editors

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Guest Editor
1. Department of Basic and Preclinical Sciences, Nicolaus Copernicus University in Torun, Lwowska 1, 87-100 Torun, Poland
2. Department of Animal Anatomy, Poznan University of Life Sciences, Wojska Polskiego 71C, 60-625 Poznan, Poland
Interests: arteries; carotid arteries; vascular diseases; maxillary artery; basilar artery; blood; ruminants; ruminant

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Guest Editor
1. Head of Division of Anatomy, Department of Human Morphology and Embryology, Wrocław Medical University, 50-367 Wrocław, Poland
2. Institute of Veterinary Medicine; Nicolaus Copernicus University, 87-100 Torun, Poland
3. Physiology Graduate Faculty, North Carolina State University, Raleigh, NC 27695, USA
4. Center of Assisted Reproduction, Department of Obstetrics and Gynecology, University Hospital and Masaryk University, 601 77 Brno, Czech Republic
Interests: molecular biology; primary culture; long term culture; stem cells; microarray; in vitro maturation; development; proloferation

Special Issue Information

Dear Colleagues,

We are pleased to announce the launch of “Cardiovascular Disease of Animals: Pathophysiology, Anatomy, Diagnosis and Treatment”, a Special Issue of the journal Applied Sciences.

Veterinary medicine is a vast field of science that constantly faces new challenges. The development of veterinary cardiology, especially in the field of small animals, is related to the increasing expectations of pet owners regarding the diagnosis and treatment of their pets. This is made possible by the broader use of modern diagnostic techniques, which are becoming increasingly common. Numerous scientific studies are being conducted on experimental animals directly related to using the results in human medicine.

In this Special Issue, we encourage everyone to share the results of their studies in pathophysiology, pathomorphology, anatomy, diagnosis and treatment of animals’ cardiovascular diseases.

In this Special Issue, “Cardiovascular Disease of Animals: Pathophysiology, Anatomy, Diagnosis and Treatment”, original research articles and reviews are welcome. Research areas may include (but are not limited to) the following:

  • Angiology;
  • Animal anatomy;
  • Cardiology;
  • Cardiovascular diseases;
  • Congenital heart diseases; 
  • Diagnostics in cardiovascular diseases;
  • Echocardiography;
  • Heart diseases;
  • Image techniques;
  • Pathological anatomy;
  • Pathophysiology;
  • Teaching;
  • Treatments for cardiovascular diseases.

Dr. Maciej Zdun
Prof. Dr. Bartosz Kempisty
Guest Editors

Manuscript Submission Information

Manuscripts should be submitted online at by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Applied Sciences is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2400 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.


  • anatomy
  • pathological anatomy
  • pathophysiology
  • diagnosis
  • treatment
  • heart
  • vessels
  • blood

Published Papers (1 paper)

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15 pages, 835 KiB  
Pulmonary Hypertension Secondary to Myxomatous Mitral Valve Disease in Dogs: Current Insights into the Histological Manifestation and Its Determining Factors
by Arkadiusz Grzeczka, Urszula Pasławska, Szymon Graczyk, Paulina Antosik, Marcin Zawadzki and Robert Pasławski
Appl. Sci. 2024, 14(6), 2577; - 19 Mar 2024
Viewed by 654
Pulmonary venous hypertension (PVH) is caused by deteriorating left ventricular function. The most common cause of PVH in dogs is myxomatous mitral valve degeneration (MMVD). It causes left ventricular volume overload and an increase in left atrial and pulmonary venous pressure (PVH), which [...] Read more.
Pulmonary venous hypertension (PVH) is caused by deteriorating left ventricular function. The most common cause of PVH in dogs is myxomatous mitral valve degeneration (MMVD). It causes left ventricular volume overload and an increase in left atrial and pulmonary venous pressure (PVH), which leads to pulmonary vascular wall remodeling and contributes to the perpetuation and worsening of PVH. Pulmonary vascular wall remodeling is also characteristic of pulmonary arterial hypertension (PAH). However, the changes in PVH arise secondary to heart failure and vascular remodeling progresses as the disease progresses. On the other hand, PAH is a primary disease that can be triggered, for example, by the use of certain drugs. Similar structural changes may suggest the influence of similar pathophysiological mechanisms or the intermediation of similar mediators. Therefore, this article discusses recent and hitherto uncommented findings elucidating the pathophysiology of the processes and influences on the pattern of histological changes observed in pulmonary hypertension secondary to degenerative mitral valve disease. In particular, we focus on the activity of factors such as endothelin, serotonin, and nitric oxide, which are involved in pulmonary vascular wall remodeling in both PVH and PAH. Full article
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