Trace Elements, Redox Balance, and Neurological Diseases

A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".

Deadline for manuscript submissions: 31 October 2024 | Viewed by 22

Special Issue Editors


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Guest Editor
Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang 050024, China
Interests: iron metabolism; iron misregulation; redox imbalance; Parkinson's disease; Alzheimer's disease; stroke; mental and emotional disorders; preparation and safety evaluation of brain-targeted nanomedicines
Special Issues, Collections and Topics in MDPI journals

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Guest Editor
School of Health and Life Sciences, University of Health and Rehabilitation Sciences, Qingdao, China
Interests: neurodegenerative diseases; Parkinson’s disease; α-synuclein; iron metabolism; proteostasis; ghrelin

Special Issue Information

Dear Colleagues,

Trace elements are indispensable for organisms to execute normal physiological functions. In recent years, breakthroughs have been achieved in the exploration of the following areas: imbalanced trace element metabolism has a significant impact on the onset of neurological diseases; the intricate signaling pathways that initiate redox imbalance are closely related to trace elements; oxidation-reduction imbalance plays an important role in the genesis and progression of neurological diseases and the aging process. Therefore, the demand to focus on showcasing recent advances in these areas is becoming increasingly strong.

This Special Issue aims to explore the latest progress made in terms of the effects of trace elements such as iron, copper, zinc, selenium, manganese, etc., on neurodevelopment, aging, psychological disorders, and neurological diseases, including stroke, Parkinson's disease, Alzheimer's disease, Huntington's disease, ALS, etc., and to explore the mechanisms of cell division, differentiation, migration, aging, apoptosis, ferroptosis, and necrosis related to the regulation of redox signaling caused by an imbalance in trace element metabolism.

This Special Issue will present new advances in research in this field, promoting further development of the study of trace elements, redox balance, and neurological diseases.

Prof. Dr. Yan-Zhong Chang
Prof. Dr. Hong Jiang
Guest Editors

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Antioxidants is an international peer-reviewed open access monthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • iron
  • copper
  • zinc
  • selenium
  • redox
  • Alzheimer's disease
  • Parkinson's disease
  • stroke
  • ageing
  • brain-targeted nanomedicines

Published Papers

This special issue is now open for submission.
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