Cardiorenal Syndrome: Pathophysiology, Diagnosis, and Treatment
A special issue of Journal of Clinical Medicine (ISSN 2077-0383). This special issue belongs to the section "Cardiology".
Deadline for manuscript submissions: closed (25 February 2024) | Viewed by 167
Special Issue Editor
2. Cardiac Intensive Care Unit, Tel Aviv Sourasky Medical Center, Tel Aviv 62431, Israel
Interests: cardiorenal syndromes; acute renal damage; heart failure
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
A large proportion of patients admitted to hospitals have various degrees of heart and kidney dysfunction. A diseased heart has numerous negative effects on kidney function but, at the same time, renal insufficiency can significantly impair cardiac function. Thus, the direct and indirect effects of each organ that is dysfunctional can initiate and perpetuate the combined disorder of the two organs through a complex combination of neurohormonal feedback mechanisms. Primary disorders of one of these two organs often result in secondary dysfunction or injury to the other. Such interactions represent the pathophysiological basis for a clinical entity called cardiorenal syndrome (CRS). Although generally defined as a condition characterized by the initiation and/or progression of renal insufficiency secondary to heart failure, the term CRS is also used to describe the negative effects of reduced renal function on the heart and circulation. Recent advances in clinical sciences have improved our understanding of organ crosstalk and have demonstrated the efficacy of some therapies in attenuating both cardiac and renal injury.
The aim of this Special Issue is to present clinical research that improves our understanding of these complex and important organ interactions. Original research articles and systematic and comprehensive review papers are welcome.
Dr. Yacov Shacham
Guest Editor
Manuscript Submission Information
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Keywords
- acute kidney injury
- biomarkers
- serum creatinine
- tubular damage
- glomerular filtration rate
- heart failure
