Genetics and Epigenetics of Breast Cancer: A Topic in Evolution
A special issue of Genes (ISSN 2073-4425). This special issue belongs to the section "Molecular Genetics and Genomics".
Deadline for manuscript submissions: closed (20 July 2023) | Viewed by 276
Special Issue Editors
Interests: epigenetic markers; breast cancer; metastasis; tumor suppressor genes
Special Issue Information
Dear Colleagues,
As we enter a new era of precision medicine, our knowledge of the relationships between genomic, epigenomic, transcriptomic, and molecular factors and cancer development is increasing every year.
Breast cancer is a heterogeneous disease that is challenging to characterize and treat. The explosion of genetic and epigenetic research in recent years has helped in understanding the complexity of breast cancer’s genetic landscape both at the onset of the disease and during tumor progression. There are many options for treating hereditary breast cancers. Hereditary breast cancers are different from sporadic cancers in ways that can affect treatment choices. Genetic test results may influence some treatment decisions. The choice of treatment can be personalized based on cancer type, stage, and genetics.
In this Special Issue, original research articles and reviews are welcome. Research areas may include but are not limited to diverse aspects of breast cancer research, from susceptibility to new targets to therapy.
Prof. Dr. Abdul Rauf Shakoori
Dr. Muhammad Usman Rashid
Guest Editors
Manuscript Submission Information
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Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2600 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- breast cancer risk
- metastases
- genetic landscape
- diagnostic/prognostic/predictive biomarkers
- next-generation sequencing
- epithelial mesenchymal transition (EMT)
- tumor suppressor genes
