Special Issue "The Biology of Non-Coding RNAs and Metabolic Diseases"
A special issue of Non-Coding RNA (ISSN 2311-553X).
Deadline for manuscript submissions: closed (31 December 2016)
2. Department of Diabetes, Central Clinical School, Monash University, Melbourne, VIC 3004, Australia
Interests: diabetes; clinical and molecular epigenetics; chromatin; DNA and RNA methylation; histones; transcriptional regulation; computational epigenomics; metabolic disease
Special Issues, Collections and Topics in MDPI journals
Non-Coding RNA is now accepting submissions for a special issue on the biology of ncRNAs and metabolism and its disorders. The special issue is co-guest-edited by the Professor Assam El-Osta from the Baker IDI Heart & Diabetes Institute, and Professor Ronald Ma from The Chinese University of Hong Kong. This Special Issue will also include commissioned topical reviews written by leaders in the field. Accepted papers are published online immediately after copy editing.
Non-Coding RNA is an Open Access journal. There is no Article Processing Charges (APCs) for papers submitted in 2016.
The rising global epidemic of obesity, cardiovascular disease and diabetes underscores the current challenge of characterizing the metabolically responsive epigenome and understanding the role and interaction of ncRNAs. For example, studies in recent years investigating the molecular mechanisms linking fat and glucose metabolism with transcription highlight important regulatory determinants that underpin the expression of genes implicated in metabolic disease which are considered critical yet remain poorly understood. Together with the expression and interactions of ncRNAs, increasing evidence indicates that altered metabolism contributes to disease.
We will consider Research, Methods and Software manuscripts of exceptional interest on the following topics:
- Discovery of ncRNAs implicated in metabolic health and disease
- Overlap of epigenomics and ncRNAs in metabolism
- Pre-clinical and clinical studies investigating metabolic or hyperglycemic memory
- Integrative and multi-omic approaches to study metabolic disease
- Population studies providing insights into ncRNAs
- The use of ncRNAs and genomics in model organisms of metabolic disease
- Connection between the nutritional environment of the developing fetus and ncRNA changes underlying pathological gene expression
- Dietary factors conferring epigenetic or ncRNA changes associated with metabolic dysfunction
- Resources of exceptional interest to the study of metabolism and its disorders
Prof. Dr. Assam El-Osta
Prof. Dr. Ronald Ma
Manuscript Submission Information
Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.
Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Non-Coding RNA is an international peer-reviewed open access semimonthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 1600 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.