Cancer Cell Proliferation
A special issue of Cancers (ISSN 2072-6694).
Deadline for manuscript submissions: closed (31 March 2015) | Viewed by 75630
2. Department of Medicine, University of Oklahoma, Oklahoma City, OK 73126, USA
Interests: Immunotherapy; CAR-T cells; tumor microenvironment; checkpoint protein; hypoxia; tumor survival signaling
Special Issues, Collections and Topics in MDPI journals
Cancer cell growth and proliferation can be activated by extracellular growth factors that bind to tyrosine kinase receptors or by extracellular matrix-integrin-focal adhesion kinase, Src signaling and its down-stream signaling pathways. In many cancer types, abnormal regulation of tyrosine kinase receptor signaling (TKR)s plays a role in cell cycle, proliferation, survival and oncogenesis. Targeting cancer cell proliferation by inhibiting TKRs, integrins and its down-stream intracellular signaling pathways is a potential approach to development of anticancer targeted therapy.
Dr. Vita Golubovskaya
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- growth factor receptors
- tyrosine kinase
- intracellular survival signaling
- focal adhesion kinase