Astrocytes in Neurodegenerative Diseases
A special issue of Brain Sciences (ISSN 2076-3425). This special issue belongs to the section "Neurodegenerative Diseases".
Deadline for manuscript submissions: closed (25 February 2024) | Viewed by 171
Special Issue Editor
Interests: astrocyte; receptor; signalling; noradrenaline; G-protein coupled receptors; glioblastoma; imaging; patch clamp; neuroprotection; optogenetics; Ca2+; cAMP; brainstem; cardio-respiratory control
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The term “neurodegenerative disease” usually refers to conditions such as Alzheimer’s disease, Huntington’s disease, various ataxias and a vast number of rare genetic diseases that lead to progressive loss of neurons, loss of cognitive and motor functions and, in some cases, death. However, signs of neurodegeneration are also evident in disorders such as epilepsy and depression, which are not usually listed under this title. Although the term “neurodegeneration” obviously implies a loss of viability of neurons, it is now generally acknowledged that astrocytes are intimately involved in any such process. We know that astrocytes aid the resilience of neurons by various means. On the other hand, some studies suggest that under certain conditions, they may actually be a part of the pathological mechanism.
In this Special Issue, we are seeking to publish studies looking at the interplay between astrocytes and neurones in all pathological states where the endpoint is neurodegeneration. Mechanisms by which astrocytes protect neurons from damage, the cooperation of astrocytes with microglia in dealing with degenerating neurons, and the plasticity of astrocytes in disease are potential topics for papers in this volume. Studies demonstrating pathological mechanisms linked to processes within astrocytes are also welcome.
Prof. Dr. Sergey Kasparov
Guest Editor
Manuscript Submission Information
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Keywords
- astrocytes
- neurodegeneration
- Alzheimer disease
- Huntington disease
- signaling
- neuroprotection
